Rheumatoid arthritis and Proteus by Alan Ebringer

By Alan Ebringer

Rheumatoid Arthritis and Proteus explores the concept that Rheumatoid arthritis is attributable to a urinary tract an infection because of Proteus micro organism. Rheumatoid arthritis is a critical, painful and crippling disorder affecting hundreds of thousands of individuals during the international, particularly girls. Genetic reviews during the last 30 years have proven that people who own the white cellphone blood teams HLA-DR1/4 hold a susceptibility series and usually tend to strengthen the disorder. This publication makes use of the tools of Sir Karl Popper, the thinker of technology, to provide 12 “Popper sequences” that have been pointed out to point that Proteus is the causative agent of Rheumatoid Arthritis. Rheumatoid Arthritis and Proteus proposes that Anti-Proteus cures will be as early as attainable to avoid the crippling and irreversible joint deformities that take place in Rheumatoid Arthritis.

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During his first year residency, the author of this book witnessed a spectacular case of a 16-year-old female rheumatic fever patient with a pronounced heart murmur and severe chorea which prevented her from walking or going to the toilet and the attending senior physician made the confident claim that he would cure her in 1 week with the use of penicillin antibiotics. On the subsequent ward-round, 1 week later, the patient was running around filling the water bottles of the other elderly stroke female patients in the ward whose clinical status had not changed.

If rheumatic fever could be caused by cross-reactive autologous antibodies evoked by an infection, why not other diseases? Conclusions Longitudinal studies are required in rheumatoid arthritis patients to assess whether antibiotic intervention will reduce the anti-Proteus antibody titre and modify the clinical outcome of the arthritic disease. The role of Proteus in rheumatoid arthritis patients merits further study. References Bennet PH, Wood PNH. Population studies of the rheumatic diseases. Amsterdam: Excerpta Medica; 1968.

1958). Ten rheumatoid arthritis patients were Enzyme-Linked Immunosorbent Assay (ELISA) 37 treated with gold sodium thiomalate, each patient receiving a cumulative dose of 1 g. Nineteen rheumatoid arthritis patients received lymphoid irradiation therapy (Hanly et al. 1986). These were randomly chosen to receive total doses of either 750 rad (9 patients) or 2,000 rad (10 patients) lymphoid irradiation. Radiotherapy was given on an outpatient basis four times weekly, using an 8 meV linear accelerator.

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